Persistent oligemia of rat cerebral cortex in the wake of spreading depression.

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Standard

Persistent oligemia of rat cerebral cortex in the wake of spreading depression. / Lauritzen, M; Jørgensen, M B; Diemer, N H; Gjedde, A; Hansen, A J.

In: Annals of Neurology, Vol. 12, No. 5, 1982, p. 469-74.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Lauritzen, M, Jørgensen, MB, Diemer, NH, Gjedde, A & Hansen, AJ 1982, 'Persistent oligemia of rat cerebral cortex in the wake of spreading depression.', Annals of Neurology, vol. 12, no. 5, pp. 469-74. https://doi.org/10.1002/ana.410120510

APA

Lauritzen, M., Jørgensen, M. B., Diemer, N. H., Gjedde, A., & Hansen, A. J. (1982). Persistent oligemia of rat cerebral cortex in the wake of spreading depression. Annals of Neurology, 12(5), 469-74. https://doi.org/10.1002/ana.410120510

Vancouver

Lauritzen M, Jørgensen MB, Diemer NH, Gjedde A, Hansen AJ. Persistent oligemia of rat cerebral cortex in the wake of spreading depression. Annals of Neurology. 1982;12(5):469-74. https://doi.org/10.1002/ana.410120510

Author

Lauritzen, M ; Jørgensen, M B ; Diemer, N H ; Gjedde, A ; Hansen, A J. / Persistent oligemia of rat cerebral cortex in the wake of spreading depression. In: Annals of Neurology. 1982 ; Vol. 12, No. 5. pp. 469-74.

Bibtex

@article{08867c10b31511debc73000ea68e967b,
title = "Persistent oligemia of rat cerebral cortex in the wake of spreading depression.",
abstract = "Cerebral blood flow during and after spreading depression (SD) was studied in rat brain by quantitative autoradiography. The rise of cortical blood flow during SD was followed by 20 to 25% reduction below normal after SD, lasting at least an hour. Blood flow in th putamen, thalamus, and hippocampus did not change at any time during or after SD. Previous measurements of cerebral blood flow in humans showed that migraine attacks may be accompanied by wavelike spreading oligemia (Olesen et al, 1981). We speculate that the spreading oligemia of migraine may be a phenomenon physiologically related to the present finding of an oligemia after SD.",
author = "M Lauritzen and J{\o}rgensen, {M B} and Diemer, {N H} and A Gjedde and Hansen, {A J}",
year = "1982",
doi = "10.1002/ana.410120510",
language = "English",
volume = "12",
pages = "469--74",
journal = "Annals of Neurology",
issn = "0364-5134",
publisher = "JohnWiley & Sons, Inc.",
number = "5",

}

RIS

TY - JOUR

T1 - Persistent oligemia of rat cerebral cortex in the wake of spreading depression.

AU - Lauritzen, M

AU - Jørgensen, M B

AU - Diemer, N H

AU - Gjedde, A

AU - Hansen, A J

PY - 1982

Y1 - 1982

N2 - Cerebral blood flow during and after spreading depression (SD) was studied in rat brain by quantitative autoradiography. The rise of cortical blood flow during SD was followed by 20 to 25% reduction below normal after SD, lasting at least an hour. Blood flow in th putamen, thalamus, and hippocampus did not change at any time during or after SD. Previous measurements of cerebral blood flow in humans showed that migraine attacks may be accompanied by wavelike spreading oligemia (Olesen et al, 1981). We speculate that the spreading oligemia of migraine may be a phenomenon physiologically related to the present finding of an oligemia after SD.

AB - Cerebral blood flow during and after spreading depression (SD) was studied in rat brain by quantitative autoradiography. The rise of cortical blood flow during SD was followed by 20 to 25% reduction below normal after SD, lasting at least an hour. Blood flow in th putamen, thalamus, and hippocampus did not change at any time during or after SD. Previous measurements of cerebral blood flow in humans showed that migraine attacks may be accompanied by wavelike spreading oligemia (Olesen et al, 1981). We speculate that the spreading oligemia of migraine may be a phenomenon physiologically related to the present finding of an oligemia after SD.

U2 - 10.1002/ana.410120510

DO - 10.1002/ana.410120510

M3 - Journal article

C2 - 7181451

VL - 12

SP - 469

EP - 474

JO - Annals of Neurology

JF - Annals of Neurology

SN - 0364-5134

IS - 5

ER -

ID: 14943664