Pathophysiology of the human brain after stroke, monitored by positron emission tomography.

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Pathophysiology of the human brain after stroke, monitored by positron emission tomography. / Gjedde, A.

In: Experimental Pathology, Vol. 42, No. 4, 1991, p. 221-7.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Gjedde, A 1991, 'Pathophysiology of the human brain after stroke, monitored by positron emission tomography.', Experimental Pathology, vol. 42, no. 4, pp. 221-7.

APA

Gjedde, A. (1991). Pathophysiology of the human brain after stroke, monitored by positron emission tomography. Experimental Pathology, 42(4), 221-7.

Vancouver

Gjedde A. Pathophysiology of the human brain after stroke, monitored by positron emission tomography. Experimental Pathology. 1991;42(4):221-7.

Author

Gjedde, A. / Pathophysiology of the human brain after stroke, monitored by positron emission tomography. In: Experimental Pathology. 1991 ; Vol. 42, No. 4. pp. 221-7.

Bibtex

@article{425e3950b31511debc73000ea68e967b,
title = "Pathophysiology of the human brain after stroke, monitored by positron emission tomography.",
abstract = "Six stroke patients had positron tomograms both in the acute stage of the cerebrovascular accident (16 to 38 h after onset), and one week later. In and around the infarct, the studies revealed a wide range of metabolic states. In the healthy regions of the brain, all measured physiological variables, including the density of capillaries that transported glucose, blood flow, and oxygen and glucose metabolism, changed in parallel (recruitment). In the regions suffering the consequences of stroke, in the second study, the physiological couple between capillary density, metabolism, and flow was significantly impaired, and the impairment was proportional to the severity of ischemia in the first study. The research report of these findings appeared in the Journal of Cerebral Blood Flow and Metabolism (Gjedde et al. 1990).",
author = "A Gjedde",
year = "1991",
language = "English",
volume = "42",
pages = "221--7",
journal = "Experimental Pathology",
issn = "0232-1513",
publisher = "Elsevier",
number = "4",

}

RIS

TY - JOUR

T1 - Pathophysiology of the human brain after stroke, monitored by positron emission tomography.

AU - Gjedde, A

PY - 1991

Y1 - 1991

N2 - Six stroke patients had positron tomograms both in the acute stage of the cerebrovascular accident (16 to 38 h after onset), and one week later. In and around the infarct, the studies revealed a wide range of metabolic states. In the healthy regions of the brain, all measured physiological variables, including the density of capillaries that transported glucose, blood flow, and oxygen and glucose metabolism, changed in parallel (recruitment). In the regions suffering the consequences of stroke, in the second study, the physiological couple between capillary density, metabolism, and flow was significantly impaired, and the impairment was proportional to the severity of ischemia in the first study. The research report of these findings appeared in the Journal of Cerebral Blood Flow and Metabolism (Gjedde et al. 1990).

AB - Six stroke patients had positron tomograms both in the acute stage of the cerebrovascular accident (16 to 38 h after onset), and one week later. In and around the infarct, the studies revealed a wide range of metabolic states. In the healthy regions of the brain, all measured physiological variables, including the density of capillaries that transported glucose, blood flow, and oxygen and glucose metabolism, changed in parallel (recruitment). In the regions suffering the consequences of stroke, in the second study, the physiological couple between capillary density, metabolism, and flow was significantly impaired, and the impairment was proportional to the severity of ischemia in the first study. The research report of these findings appeared in the Journal of Cerebral Blood Flow and Metabolism (Gjedde et al. 1990).

M3 - Journal article

C2 - 1959582

VL - 42

SP - 221

EP - 227

JO - Experimental Pathology

JF - Experimental Pathology

SN - 0232-1513

IS - 4

ER -

ID: 14946795