Parkinson's disease and mitochondrial gene variations: A review

Research output: Contribution to journal › Journal article › Research › peer-review

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Parkinson's disease and mitochondrial gene variations : A review. / Andalib, Sasan; Vafaee, Manouchehr Seyedi; Gjedde, Albert.

In: Journal of the Neurological Sciences, Vol. 346, No. 1-2, 07.08.2014, p. 11-19.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Andalib, S, Vafaee, MS & Gjedde, A 2014, 'Parkinson's disease and mitochondrial gene variations: A review', Journal of the Neurological Sciences, vol. 346, no. 1-2, pp. 11-19. https://doi.org/10.1016/j.jns.2014.07.067

APA

Andalib, S., Vafaee, M. S., & Gjedde, A. (2014). Parkinson's disease and mitochondrial gene variations: A review. Journal of the Neurological Sciences, 346(1-2), 11-19. https://doi.org/10.1016/j.jns.2014.07.067

Vancouver

Andalib S, Vafaee MS, Gjedde A. Parkinson's disease and mitochondrial gene variations: A review. Journal of the Neurological Sciences. 2014 Aug 7;346(1-2):11-19. https://doi.org/10.1016/j.jns.2014.07.067

Author

Andalib, Sasan ; Vafaee, Manouchehr Seyedi ; Gjedde, Albert. / Parkinson's disease and mitochondrial gene variations : A review. In: Journal of the Neurological Sciences. 2014 ; Vol. 346, No. 1-2. pp. 11-19.

Bibtex

@article{034a905b25c64cc097c97d28551664f0,
title = "Parkinson's disease and mitochondrial gene variations: A review",
abstract = "Parkinson's disease (PD) is a common disorder of the central nervous system in the elderly. The pathogenesis of PD is a complex process, with genetics as an important contributing factor. This factor may stem from mitochondrial gene variations and mutations as well as from nuclear gene variations and mutations. More recently, a particular role of mitochondrial dysfunction has been suggested, arising from mitochondrial DNA variations or acquired mutations in PD pathogenesis. The present review summarizes and weighs the evidence in support of mitochondrial DNA (mtDNA) variations as important contributors to the development and course of PD.",
author = "Sasan Andalib and Vafaee, {Manouchehr Seyedi} and Albert Gjedde",
note = "Copyright {\textcopyright} 2014 Elsevier B.V. All rights reserved.",
year = "2014",
month = aug,
day = "7",
doi = "10.1016/j.jns.2014.07.067",
language = "English",
volume = "346",
pages = "11--19",
journal = "Journal of the Neurological Sciences",
issn = "0022-510X",
publisher = "Elsevier",
number = "1-2",

}

RIS

TY - JOUR

T1 - Parkinson's disease and mitochondrial gene variations

T2 - A review

AU - Andalib, Sasan

AU - Vafaee, Manouchehr Seyedi

AU - Gjedde, Albert

N1 - Copyright © 2014 Elsevier B.V. All rights reserved.

PY - 2014/8/7

Y1 - 2014/8/7

N2 - Parkinson's disease (PD) is a common disorder of the central nervous system in the elderly. The pathogenesis of PD is a complex process, with genetics as an important contributing factor. This factor may stem from mitochondrial gene variations and mutations as well as from nuclear gene variations and mutations. More recently, a particular role of mitochondrial dysfunction has been suggested, arising from mitochondrial DNA variations or acquired mutations in PD pathogenesis. The present review summarizes and weighs the evidence in support of mitochondrial DNA (mtDNA) variations as important contributors to the development and course of PD.

AB - Parkinson's disease (PD) is a common disorder of the central nervous system in the elderly. The pathogenesis of PD is a complex process, with genetics as an important contributing factor. This factor may stem from mitochondrial gene variations and mutations as well as from nuclear gene variations and mutations. More recently, a particular role of mitochondrial dysfunction has been suggested, arising from mitochondrial DNA variations or acquired mutations in PD pathogenesis. The present review summarizes and weighs the evidence in support of mitochondrial DNA (mtDNA) variations as important contributors to the development and course of PD.

U2 - 10.1016/j.jns.2014.07.067

DO - 10.1016/j.jns.2014.07.067

M3 - Journal article

C2 - 25151610

VL - 346

SP - 11

EP - 19

JO - Journal of the Neurological Sciences

JF - Journal of the Neurological Sciences

SN - 0022-510X

IS - 1-2

ER -

ID: 126434970